Pathophysiology of Eating Disorders Essay Sample

Pathophysiology of Eating Disorders

Eating disorders are conditions portrayed by severe disturbances in ones eating behavior together with extreme anxiety over body weight.  They present themselves in varied manner though in most cases they appear with harsh medical comorbidity. There are two major classifications of eating disorders including anorexia nervosa and bulimia nervosa with many patients indicating a combination of both. In pathophysiology of the disorders, both psychosocial and biologic factors are implicated (Patricia, 1994)

Psychosocial factors

Michelle et al (2001) state that in families of children with eating disorders, soaring levels of chaos, hostilities, isolation, empathy and low level of nurturance are reported. Anorexia is postulated as a reaction to demands on adolescents to independently behave or as a response to societal pressure to be lean. Patients with anorexia nervosa normally, are high achievers. It is estimated that two thirds of them live with their parents. It is also viewed by many that their bodies are under their parent’s control although solely, family dynamics do not cause anorexia nervosa. Those with bulimia nervosa are depicted to have impulse regulation difficulties.

Biologic factors

Denial of hunger in patients with anorexia nervosa might be as a result of endogenous opioids. Dieting is thought to increase eating disorders developing risk. After purging, patients with bulimia nervosa have been described with increased levels of endorphin which is likely to induce well being feelings. Reduced norepinephrine turnover and activity are implied by low 3-methoxy-4-hydroxyphenylglco levels in the anorexia nervosa patient’s cerebrospinal fluid and urine. Bulimia nervosa patients often gain from antidepressants as they sustain a pathophysiologic role for norepinephrine and serotonin.

Starvation leads to several biochemical changes like thyroid function’s suppression, amenorrhea, hypercortisolemia and non suppression of dexamethasone. Additionally, anorexia risk may rise with a polymorphism of the promoter region of serotonin 2a receptor. Polymorphisms in some genes also play a role at the melancortin. Excessive secretion of ghrelin is experienced in bulimia nervosa. Ghrelin receptor gene polymorphism has been linked with both Prader-Willi syndrome and hyperphagia of bulimia.

Share this article

  1. Harmful Effects of BPA
  2. Forensic Psychologist
  3. Health Care Management
  4. The Binary Constructions of Sex, Gender and Sexuality
  5. Anorexia and eating disorders
  6. Productive and Counterproductive Behaviors
  7. The Role of Nursing in Patients with Obstructive Sleep Apnea
  8. Maternal Health & Pediatrics: Rheumatic Heart Disease
  9. Report on nutrition practices among ethnic groups in the U.S
  10. Marijuana and Medicine
 

What Our Customers Say

 

Why us

Experienced writers with
the highest satisfaction rates.

Great discounts for new
customers and returned ones.

Full confidentiality of your
personal and contact information.

Our stats

Preparing Orders  

0

Active Writers  

0

Support Agents  

0

 

Receive 10% discount

and rock this semester!

Now Accepting Apple Pay!
Use discount code first10 Get 10% OFF Your First Order!
X
Online - please click here to chat